Subtitle The Transporter
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subtitle The Transporter
About Us The Hazardous Waste Compliance and Enforcement program administers the RCRA (Resource Conservation Recovery Act) program in New Jersey. There is also a transportation oversight function that includes, in coordination with the State Police, roadside operations and hazardous waste transporter terminal audits. What We Do The Hazardous Waste Compliance and Enforcement program performs the following activities to ensure that hazardous waste is properly identified and collected, transported, treated and disposed of in an environmentally sound manner:
In 1885, Merrit became Maffet's partner. As the circulation of the Transporter approached 1,000, it acquired the subtitle, "Indian and Stock Journal." The paper opposed opening the territory for white settlement and called on the government to protect the interests of Indians and the cattle industry. That same year, hostilities broke out between the Indians and newly arriving Boomers. U.S. Army Captain Jesse Matlock Lee temporarily commandeered the agency, started an investigation and expelled many of the illegal settlers. Businesses in the territory were thereafter required to furnish approved bonds to obtain an operating license from the Department of the Interior. The Cheyenne Transporter presented a $10,000 bond and obtained a license; however, within a year the newspaper was suspended due to Indian unrest following Captain Lee's departure. Publication never resumed, making August 12, 1886, the final issue of the Transporter.
Hazardous waste transporters are individuals or entities that move hazardous waste from one site to another by highway, rail, water, or air. Hazardous waste transporters play an integral role in the hazardous waste management system by delivering hazardous waste from its point of generation to ultimate destination. This includes transporting hazardous waste from a generator's site to a facility that can recycle, treat, store or dispose of the waste. It can also include transporting treated hazardous waste to a site for further treatment or disposal.
Because hazardous waste transporters move regulated wastes on public roads, highways, rails, and waterways, EPA and the U.S. Department of Transportation (U.S. DOT) jointly developed the hazardous waste transporter regulations.
With the exception of water and rail shipments, a copy of the manifest must accompany a copy of the shipment of waste at all times. Once a transporter accepts a waste, the transporter is required to deliver the entire quantity of waste to the next designated transporter or facility. When the waste arrives at its next destination, the transporter must have the manifest signed and dated by the recipient. The transporter must keep a copy of the manifest for three years.
Water and rail transporters must comply with the directions on the manifest, obtain an EPA ID number, and be listed on the manifest like highway and air shipments. However, the manifest is not required to physically accompany these shipments at all times. Instead, water and rail transporters can use another shipping document instead of the manifest, provided that it contains the same information as the manifest. Additionally,
A special exemption from the manifest requirements exists for transporters who handle certain recycled (or reclaimed) wastes generated by SQGs (See 40 CFR section 263.20 (h)). This exemption is intended to facilitate the recycling of small quantities of hazardous wastes that are transported in a protective manner.
If a transporter discharges or spills hazardous waste, he or she is required to take appropriate, immediate action to protection human health and the environment such as notifying local authorities or diking the discharge area. Additionally:
To avoid discrepancies and redundant regulations, the hazardous waste transporter regulations adopted portions of the U.S. DOT regulations for the safe transport of DOT classified hazardous materials. The DOT references include requirements for labeling, marking, placarding, and containers, and the DOT requirements referenced above for responding to spills. Transporters of hazardous waste should consult and comply with all applicable requirements in the U.S. DOT regulations.
Transporters accepting hazardous waste from a generator or another transporter may need to hold waste temporarily during the normal course of transportation. A transfer facility is defined as any transportation-related facility, such as loading docks, parking areas, storage areas, and other similar areas where shipments are temporarily held. A hazardous waste transporter may hold waste without a storage permit in containers at a transfer facility for 10 days or less as long as the waste is manifested and kept in U.S. DOT specification containers. Storage in stationary containers is prohibited unless the transfer facility has a RCRA permit or interim status.
If a transporter stores waste in containers at a transfer facility for more than 10 days, the transfer facility becomes a storage facility subject to all applicable requirements for treatment, storage and disposal facilities (TSDFs).
The regulations governing imports and exports of hazardous waste are primarily found in 40 CFR part 262, subpart E, the section for hazardous waste generators. However, transporters are required to comply with these regulations if they import hazardous waste into the United States.
Mississippi law requires each generator of greater than 220 pounds of hazardous waste in any calendar month, each transporter of hazardous waste, and the owner or operator of any facility for the treatment, storage, recycling or disposal of hazardous waste to report annually the types and amounts of hazardous waste generated, managed and/or shipped during the preceding calendar year to the Mississippi Department of Environmental Quality (MDEQ).
This paper re-construct genome-wide analysis for identify MATE transporter in Medicago sativa L , which is an important crops, but sensitive to acidic soil condition. Author identified 88 MATE genes, which were divided 4 subfamilies comprising 11 subgroups. As a result of expression analysis by RT-PCR, they found 10 candidate MATE genes for Al tolerance mechanism in Alfalfa. I think this is beneficial data for understanding of molecular Al tolerance mechanism in Medicago sativa in future.
Figure 2; Author described G1; Al detoxification/iron translocation. It should be citrate transporter. G3; Efflux of various compounds. It is also incorrect caption. Because almost all MATE is efflux transporter. And G3 group including NtJAT1. This protein tansport nicotine, which is one of the alkaloid. Could you consider caption of G1,G2, G3, G4 in Fig 2.Line 57-59; To date, 56, 53, 117 and 70 MATE transporters have been identified from Arabidopsis thaliana (Li et al. 2002), Oryza sativa (Tiwari et al. 2014), Glycine max (Liu et al. 2016), Medicago truncatula (Wang et al. 2017), and Zea mays (Zhu et al. 2016), respectively. This number is different in Line 113-114. And the number of Zea mays is including ?Line 164-166; The ORFs of these genes 165 varied in length from 321 (MsMATE80) to 1788 (MsMATE10) bp. This length is different from your data. Supplemental data showed that MsMATE80 is 1485bp, MsMATE10 is 1449 (Sup data1).
Phosphate homeostasis is mostly regulated through humoral factors exerting direct or indirect effects on transporter proteins located in the intestine and kidney. Fibroblast growth factor 23 (FGF-23) is a major phosphate-regulating molecule, which can affect both renal and intestinal phosphate uptake to influence overall mineral ion homeostasis. We have found that Fgf-23 gene knockout mice (Fgf-23-/-) develop hyperphosphatemia that consequently leads to abnormal bone mineralization, and severe soft tissue calcifications. On the contrary, FGF-23 transgenic mice develop hypophosphatemia and produce rickets-like features in the mutant bone. Further studies using our Fgf-23 -/- mice have identified an inverse correlation between Fgf-23, and vitamin D or NaPi2a; genomic elimination of either vitamin D or NaPi2a activities from Fgf-23-/- mice could reverse severe hyperphosphatemia to hypophosphatemia, and consequently could alter skeletal mineralization, suggesting that regulation of phosphate homeostasis in Fgf-23-/- mice is vitamin D- and NaPi2a-mediated process.
N2 - Phosphate homeostasis is mostly regulated through humoral factors exerting direct or indirect effects on transporter proteins located in the intestine and kidney. Fibroblast growth factor 23 (FGF-23) is a major phosphate-regulating molecule, which can affect both renal and intestinal phosphate uptake to influence overall mineral ion homeostasis. We have found that Fgf-23 gene knockout mice (Fgf-23-/-) develop hyperphosphatemia that consequently leads to abnormal bone mineralization, and severe soft tissue calcifications. On the contrary, FGF-23 transgenic mice develop hypophosphatemia and produce rickets-like features in the mutant bone. Further studies using our Fgf-23 -/- mice have identified an inverse correlation between Fgf-23, and vitamin D or NaPi2a; genomic elimination of either vitamin D or NaPi2a activities from Fgf-23-/- mice could reverse severe hyperphosphatemia to hypophosphatemia, and consequently could alter skeletal mineralization, suggesting that regulation of phosphate homeostasis in Fgf-23-/- mice is vitamin D- and NaPi2a-mediated process.
AB - Phosphate homeostasis is mostly regulated through humoral factors exerting direct or indirect effects on transporter proteins located in the intestine and kidney. Fibroblast growth factor 23 (FGF-23) is a major phosphate-regulating molecule, which can affect both renal and intestinal phosphate uptake to influence overall mineral ion homeostasis. We have found that Fgf-23 gene knockout mice (Fgf-23-/-) develop hyperphosphatemia that consequently leads to abnormal bone mineralization, and severe soft tissue calcifications. On the contrary, FGF-23 transgenic mice develop hypophosphatemia and produce rickets-like features in the mutant bone. Further studies using our Fgf-23 -/- mice have identified an inverse correlation between Fgf-23, and vitamin D or NaPi2a; genomic elimination of either vitamin D or NaPi2a activities from Fgf-23-/- mice could reverse severe hyperphosphatemia to hypophosphatemia, and consequently could alter skeletal mineralization, suggesting that regulation of phosphate homeostasis in Fgf-23-/- mice is vitamin D- and NaPi2a-mediated process. 041b061a72